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Cerebral edema is a common challenge in the intensive care unit, and often occurs during trauma, malignant ischemic stroke, or hemorrhage. The condition describes a relative increase in the brain’s water content, as a result of pathologic fluid and solute movement between intracranial compartments. The brain’s four intracranial compartments are intracellular, extracellular, intravascular, and cerebrospinal fluid. Barriers separate each of these compartments, and allow each to maintain their own electrochemical environments. In acute brain injury, disruptions of these barriers may lead to cerebral edema formation [1,2].
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